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Deep Dive · 9 min read

Voice Disorders on the Praxis SLP: Pathology, Assessment, EBP

Voice disorders show up as differential diagnosis, instrumental assessment, and therapy selection. Here's the focused review that covers all three.

Written by The Praxis Path Editorial TeamLast verified

Cross-checked against the current ETS Praxis 5331 Speech-Language Pathology Test at a Glance and ASHA CCC-SLP standards.

Voice disorders are a Big 9 area and show up on the 5331 in three predictable ways: differential diagnosis (organic vs. functional vs. neurogenic), instrumental assessment interpretation, and evidence-based therapy selection. Here's the focused review.

Organic voice pathology — lesion identification

  • Vocal fold nodules: bilateral, symmetric, junction of anterior 1/3 and posterior 2/3. Cause: vocal abuse/misuse (yelling, throat clearing, singing without training). Common in children (teachers' kids), women aged 20–50.
  • Vocal fold polyps: usually unilateral, can be sessile or pedunculated. Cause: single traumatic event or chronic smoking. Often require surgery.
  • Vocal fold cysts: unilateral, sub-epithelial, do not resolve with voice therapy alone — require surgical excision.
  • Reinke's edema (polypoid degeneration):smoker's voice. Bilateral swelling of the superficial lamina propria. Low pitch, hoarse.
  • Contact ulcers/granulomas: posterior third, vocal process of arytenoid. Cause: reflux, intubation, hard glottal attack.
  • Laryngeal papilloma: HPV-related. Recurrent, requires surgical removal.

Neurogenic voice disorders

  • Unilateral vocal fold paralysis: CN X (recurrent laryngeal nerve) injury — often post-thyroidectomy or cardiac surgery. Breathy voice, weak cough, aspiration risk.
  • Spasmodic dysphonia (adductor): focal dystonia. Strained, strangled voice with voice breaks on vowels. First-line: Botox injections. Voice therapy supports but does not cure.
  • Spasmodic dysphonia (abductor): breathy voice breaks on voiceless consonants.
  • Parkinson's hypokinetic dysphonia: reduced loudness, monopitch, monoloudness, breathiness. First-line therapy: LSVT LOUD.
  • Essential tremor of voice: rhythmic 4–7 Hz pitch/loudness modulation, worse on sustained /a/.

Functional voice disorders

  • Muscle tension dysphonia (MTD): excessive laryngeal/paralaryngeal muscle activation without structural pathology. Voice therapy is first-line.
  • Puberphonia (mutational falsetto): post-pubertal male continues to speak in high pitch. Voice therapy highly effective.
  • Conversion aphonia: sudden whispered voice, normal cough (indicates intact structure).

Instrumental assessment — what each tool tells you

  • Videostroboscopy: gold standard for visualizing vocal fold vibration. Assesses closure, symmetry, mucosal wave, amplitude.
  • Acoustic analysis: fundamental frequency (F0), jitter (frequency perturbation), shimmer (amplitude perturbation), HNR (harmonics-to-noise ratio).
  • Aerodynamic: maximum phonation time (MPT — normal ≥ 15 sec adults), subglottal pressure, mean airflow.
  • Perceptual (CAPE-V or GRBAS): Grade, Roughness, Breathiness, Asthenia, Strain. Always paired with instrumental.

Evidence-based voice therapy approaches

  • Vocal hygiene: hydration, reduced yelling/throat clearing, reflux management. Foundation, rarely sufficient alone.
  • Resonant voice therapy (LMRVT): forward-focused, "buzzy" sensation. Good for MTD and nodules.
  • Vocal function exercises (VFEs): Stemple's 4-exercise program. Rebuilds laryngeal muscle balance.
  • LSVT LOUD: Parkinson's. High effort ("think LOUD"), 4x/week × 4 weeks, intensive.
  • SOVT (semi-occluded vocal tract) exercises: straw phonation, lip trills. Reduces impact stress, useful across many diagnoses.
  • Circumlaryngeal massage: MTD.

Referral musts

Any hoarseness > 2 weeks without obvious cause = ENT referral for laryngoscopy BEFORE voice therapy. This is an exam favorite. The SLP does not diagnose lesions; the ENT does.

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